insulin

If insulin became expensive during the war, that was only because things became more expensive in general, right?

Gratuitous insulin signaling causes fat cells to absorb glucose that could have been absorbed by a high-need muscle cell.

Do they trigger the sweetness taste receptors in the digestive system or trigger an increase in insulin?

Actually, too much insulin signaling interferes with leptin signaling, making you respond less to leptin signaling.

My article talks about one, and you seem to be an advocate for increasing insulin sensitivity, which is advocating for two.

Substituting dietary saturated fat with polyunsaturated fat changes abdominal fat distribution and improves insulin sensitivity.

It takes into account insulin spikes, metabolic damage assessments, glycogen storage, and much more.

As an insulin dependent diabetic I am consistently frustrated at how slow the medical device approval market moves.

Cells that have high need for glucose do not need much insulin signaling.

What is bad is the way it fluctuates in the body and the ability for the body to regulate glucose and insulin efficiently.

High-need muscle cells already have increased their blood glucose absorption rate due to increased energy requirements, while insulin signaling to fat cells is minimized.

There is some insulin signaling, but there is less of it.

Fear insulin resistance and glucose intolerance.” You do not state however how you would advise someone to avoid insulin resistance and glucose intolerance.

"No measure of insulin" -- insulin is not as easy to measure using off-the-shelf devices.

Statement: "improve your insulin resistance/glucose tolerance" Agreed.

Then you represent the goal of my experiment in the following way: “can I stuff enough kale in my mouth to slow down the release of insulin, and therefore reduce glucose.” This is not accurate.