oxidase

Also it's a multi-species mutation that stuck in humans and the great apes which broke the urate oxidase enzyme.

Is depletion of the glucose oxidase reagent the limiting factor, versus the ~~17 week estimated battery life?

Some companies[1] are developing reusable CGMs, the electronics are reusable, but the glucose oxidase probe, and applicator needle are still consumable.

My electrode was nanoporous platinum, which then was considered more advanced/fashionable/versatile than glucose oxidase coating.

The browning you see in cut foods is caused partly by polyphenol oxidase, an enzyme that can be activated by iron and other metals.

The non-browning apple gets that way by having one of its enzymes (polyphenol oxidase) turned off -- what could be dangerous about removing an enzyme?

So I think if I needed additional protection from oxidative damage, I'd try injecting my liver with L-gulonolactone oxidase before eating heavy fats.

It contains biologically relevant amounts of monoamine oxidase inhibitors.

Can we have another table for total content of random substances acting as monoamine oxidase A or B inhibitors for coffee, tea, soda and more?

α-Synuclein and familiar Parkinson's disease-related gene products modify the expression and activity of monoamine oxidase.

Caffeine, flavonoids and monoamine oxidase inhibitors.

Wikipedia: > It has been proposed that the loss of urate oxidase gene expression has been advantageous to hominoids, since uric acid is a powerful antioxidant and scavenger of singlet oxygen and radicals.

Presumably the papers are going to have titles like "Monoamine Oxidase Inhibition Dramatically Increases the Motivation to Self-Administer Nicotine in Rats".

Afaik from Nick Lane’s “Oxygen” the Cytochrome Oxidase made from the mitochondrial DNA have to match the Cytochrome C made from nuclear DNA.

Examples: Tea (e.g., black tea): The so-called "fermentation" is actually oxidation catalyzed by enzymes in the tea leaves themselves (like polyphenol oxidase), with little to no microbial activity.

A PEGylated form of urate oxidase, pegloticase, was FDA approved in 2010 for the treatment of chronic gout in adult patients refractory to "conventional therapy".[21] As a general rule though, you can effectively treat/prevent gout by significantly increasing consumption of water and by replacing proteins with cereal grains (or fruits and vegetables or vegetable fats).

I tried a bunch of things, like I suspected it might be DAO (Di-Amino-Oxidase) insufficiency.