lactase

We might try that (although a member of the Household is lactose-intolerant, so I'd better get the lactase capsules ready if we do).

Sure, some people don't produce lactase and can't digest it, but it's fine for the rest of us.

If you only have one copy of the gene you still produce enough lactase to fully digest milk.

But I don't know if that means I'm digesting it with lactase.

There's also some cool studies since then around the cultural migrations of lactase and lactose intolerance by Enattah and Tishkoff.

In my world, people that are lactose intolerant should just carry some lactase pills and not bother the server about it.

Tishkoff, a geneticist at the University of Pennsylvania who studies lactase persistence, told me.

Also xylitol, lactase, potassium citrate, rice bran, and vegetable glycerin are not really 'whole foods' by most definitions.

The lactase enzyme reaction is one of the simplest bits of chemistry to do.

The only adults who should specifically avoid milk are the ones without the lactase mutation, since they cannot digest the lactose in it.

The relatively late appearance (earliest known example of lactase persistence gene is from 4,300 years ago) and relatively small selective advantage (1.5%) are surprising.

It is made by introducing lactase which breaks the complex lactose down into simple galactose, and it tastes like a bowl of lucky charms.

She advises readers to “eat butter; drink milk whole, and feed it to the whole family...." Isn't lactase persistence in modern humans a consequence of pastoralism of the neolithic era?

> Multiple studies indicate that the presence of the two phenotypes "lactase persistent" and "lactase non-persistent (hypolactasia)" is genetically programmed, and that lactase persistence is not necessarily conditioned by the consumption of dairy products after the suckling period.

Lactase persistence is one of the biggest genetic changes compared to our paleolithic ancestors.

Lactase persistence isn't really the global norm.

Lactase persistence is a dominant mutation.