potentiation

It's interesting to note that aniracetam is considered an ampakine, which lowers the threshold required for long-term potentiation.

Long term potentiation also makes measuring individual neuron strength even less important- if you model potentiation correctly, then over time you'll converge accurately as understimulated connections weaken and vice versa.

Most of the rest of the brain needs time and repeated exposures to store a memory via long-term potentiation.

There is a lot of activity with DNA for long term potentiation even without this breaking/fixing stuff.

These waves are a plausible mechanism to explain the correlated potentiation of the glymphatic flow1,2 through the brain parenchyma.

Hippocampal neuronal pathways still showed the synaptic strengthening or ‘long-term potentiation’ that underlies learning and memory.

It is called Long Term Potentiation and is believed to be one of the ways information is stored in the brain.

Further evidence has shown that it may have actually been two separate potentiation mutations.

The synaptic homeostasis hypothesis (Tononi & Cirelli) proposes that sleep globally downscales synaptic strengths, renormalizing the system after a day of potentiation.

From the abstract: ‘These waves are a plausible mechanism to explain the correlated potentiation of the glymphatic flow through the brain parenchyma’.

The real issue is we have barely any clue how potentiation works and can't model it well at all.

These changes correlated with increased dendritic spine density, increased hippocampal CA1 long-term potentiation (LTP), and enhanced performance in associative and spatial learning and memory.

Long-term potentiation (LTP) "is widely considered one of the major cellular mechanisms that underlies learning and memory."[1] Spaced Repetition[2] and Spaced Learning[3] at techniques directly designed around LTP.

"Potentiation of latent mental illness" Yes, among those who are previously disposed.