microbiota

Add the fact that our gut microbiota is totally out of whack and things get ugly.

Studies in mice have show that the normal microbiota is rather static throughout the full life of the host.

Additionally, we now know that the microbiota of the soil itself can be as important as the genomes in the seeds.

This is all done through fecal microbiota transplantation commonly known as a fecal transplant.

I am pretty sure my microbiota was left in a very bad shape after some hard periods, and since then I have tried to bring it back to balance... but it seems a hard task.

I'm actually a participant in the Gardner study -- they're testing for a ton of other factors, including microbiota.

An analogy is that our GI and GI microbiota are essentially a recently discovered vital organ.

Interestingly, only a handful of species play a large role in the regulation of gut microbiota and physiology/psychology of the host.

So it would be just great to get exactly the combination of probiotics-supplements that one needs, based on this kind of uBiome test, and then just wait until you have a balanced microbiota again.

Together, these results establish that the metabolic derangements induced by NAS consumption are mediated by the intestinal microbiota.

While I'm a huge proponent of patient autonomy and knowledge, it's really, really, really a bad idea to perform fecal microbiota transplantations at home, without medical equipment or training, and with an unvetted donor.

The science comments above are interesting, but my intended take home message was NOT to promote an alternative to the tried and true methods of proper dental care, but instead draw attention to the incredible complexity of the human oral microbiota.

Here we demonstrate that consumption of commonly used NAS formulations drives the development of glucose intolerance through induction of compositional and functional alterations to the intestinal microbiota.

"The authors sum up their experimental findings by writing"In summary, our results suggest that NAS consumption in both mice and humans enhances the risk of glucose intolerance and that these adverse metabolic effects are mediated by modulation of the composition and function of the microbiota.

These NAS-mediated deleterious metabolic effects are abrogated by antibiotic treatment, and are fully transferrable to germ-free mice upon faecal transplantation of microbiota configurations from NAS-consuming mice, or of microbiota anaerobically incubated in the presence of NAS.