amyloids

And if anti-amyloids, or reducing amyloids are only a part solution.

Yes, there's in fact good evidence that the amyloids are likely an effect rather than the cause, arising from the long history of failure of drugs aimed at the amyloids.

But, if you really want an answer as to why I think there is more funding on amyloids than chronic inflammation, it is that amyloids are Rome.

It is clear that clearing the amyloids does not relieve one from the symptoms of dementia, or stop them.

But the reason people study amyloids is that they are at the very bottom of it.

What should the average person be doing to avoid accumulating amyloids in the first place?

But it’s unclear how well we can recover long-term and amyloids and biofilms are still active research areas.

The next phase of research is measuring the effect of stimulation on removal of beta-amyloids, related to Alzheimer’s prevention, concussion & TBI, and more.

We actually already have a test for the presence of amyloids - SAA blood tests commonly used to diagnose infection in horses and cats.

Misfolded proteins, amyloids, clog up your cardiovascular system until it fails.

We have other very promising medications that do nothing to amyloids.

Also Wikipedia: Pathogenic amyloids form when previously healthy proteins lose their normal structure and physiological functions (misfolding) and form fibrous deposits within and around cells.

"Amyloids" are spine-like fibrillar protein aggregates, where each vertebrate is a flattened version of a protein folded in beta-strands.

The best treatments on the market slows cognative decline measurements for up to 6 months "maybe" (could just be the result of pain relief), dates back to like 2001, and have nothing to do with amyloids.

There are many reasons anti-amyloid mABs might not work that well besides "amyloid hypothesis is bunk." You are giving them to very late-stage patients, they don't actually work that well if you look at the data, and some people also think that if you disaggregate fibers without a molecule that also inhibits new aggregation, you are just creating more "seeds" that can grow into more amyloids.

Amyloids are just one specific type of protein fold stacking on each other, and it is so, so common.