beta-amyloid

There’s been a long-running Internet movement to cast the beta-amyloid and beta-amyloid/tau hypotheses as an evil plot by scientists to siphon money away from research that works.

And then there's the persistent doubts about whether the beta-amyloid hypothesis is correct in the first place...

The non-DHA omega-3 EPA are good at preventing perivascular fibrosis and thus a better glymphatic system for the removal of beta-amyloid proteins.

You can consider the beta-amyloid fiasco as a prime example of biomed's replication issues.

Until believers in the hypothesis that beta-amyloid is critical run into crisis, alternate theories will have a hard time being funded and heard.

Most of the projects were around beta-amyloid and these grants do not stop.

But by potentially preventing our brains from flushing beta-amyloid—just by being in close proximity—it’s clear these devices already have the potential for serious damage.

There are easily hundreds of compounds that can reduce beta-amyloid in vitro.

These drugs were rooted in the fraudulent and failed beta-amyloid theory.

In a study by Johnson et al., widespread tau and beta-amyloid deposition was found in the brains of individuals who suffered a single traumatic brain injury.

Meanwhile, the false beta-amyloid theory of Alzheimers was purused for over a decade even though it was very clear informed people that it's a dumb theory.

There is a growing consensus that focusing solely on beta-amyloid has led to stagnation in treatment development, prompting researchers to explore alternative theories and treatment pathways.